1988;171:266C70. three genes, MAPK1, RAC2 and NRAS, were defined as targets from the deregulated miRNAs. NRAS and MAPK1 are known regulators of G2/M cell routine arrest. AMANTADIG treatment improved the manifestation of phosphorylated MAPK1 in 786-O cells. Subsequently, we researched the manifestation of survivin regarded PR-619 as suffering from cardiac glycosides also to regulate the … Continue reading 1988;171:266C70